WebOct 1, 2024 · A model of chronic cerebral hypoperfusion was established by ligating the common carotid arteries bilaterally in adult male rats (CAL group). Sham-operated rats underwent the same procedures ... WebORIGINAL ARTICLE L-carnitine enhances axonal plasticity and improves white-matter lesions after chronic hypoperfusion in rat brain Yuji Ueno1,2, Masato Koike3, Yoshiaki Shimada 2, Hideki Shimura1, Kenichiro Hira2, Ryota Tanaka , Yasuo Uchiyama3,4, Nobutaka Hattori2 and Takao Urabe1 Chronic cerebral hypoperfusion causes white …
Altered brain networks and connections in chronic heart failure ...
WebMar 13, 2024 · Chronic cerebral hypoperfusion (CCH), refers to a group of brain dysfunction syndromes caused by long-term decreased cerebral perfusion ( Ciacciarelli et al., 2024 ). WebApr 14, 2024 · However, the pathogenesis of chronic heart failure complicated with cognitive impairment is still unclear. One of the possible mechanisms is that chronic regional brain tissue hypoperfusion leading to functional decline in critical brain areas (Zou et al., 2024). Cognitive function involves various aspects such as attention, … flint journal obits archives
Normobaric oxygen: a novel approach for treating chronic cerebral ...
WebJun 8, 2024 · Chronic cerebral hypoperfusion (CCH) has an especially close relationship with the pathogenesis of VaD [ 1 ]. However, the mechanism of CCH-induced VaD is still unclear. Normal brain tissue needs an abundant blood supply to provide adequate energy and nutrition for the survival of nerve cells. WebIntroduction. Chronic cerebral circulation insufficiency (CCCI), which was first proposed by Japanese scholars in 1990s, is considered as a pathological status induced by persistent reduction of cerebral blood volume and flow (CBV and CBF, respectively), leading to ischemia and hypoxia in the brain tissue. 1 Long-time ischemic–hypoxic injury can cause … WebSep 16, 2024 · It has been found that the number of microglial cells is increased following cerebral hypoperfusion [8–10], and activated microglia release tumor necrosis factor (TNF)-α, interleukin (IL)-1β and IL-6, and other pro-inflammatory factors that mediate secondary brain damage. flint journal obituaries today